Alcohol Induced Cerebellar Ataxia: A Case Report

Consumption of alcohol excessively causes dependence and leads to psychological discomfort. Motivation drink is influenced by a variety neurobiological environmental variables. The proclivity an individual imbibe thought reflect balance between alcohol’s positive reinforcing (i.e., rewarding) effects, such as euphoria anxiety reduction anxiolysis), the drug’s aversive which are typically associated with negative consequences consumption (e.g., hangover or withdrawal symptoms). Neuroadaptive alterations caused continuous use abuse (such tolerance physiological dependence) regarded be critical in shift from regulated more frequent excessive, uncontrollable drinking induces cerebellar degeneration considered common type acquired toxic ataxia. Excessive midline cerebellum. Progressive trunk gait ataxia characteristic, little involvement upper limbs, eyes speech (a corollary relative hemispheric sparing). Abrupt termination after period heavy may result seizures. Generalized tonic–clonic seizures most severe form seizure this conditions. Current case report presents complaints astasia slurred few episodes diagnosis indicated Mild atrophy. Patient was advised undergo de-addiction cure. justify;">Key words: Cerebellar ataxia, Alcohol, atrophy, GABA, astasia,seizures.